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11 Conclusion In this paper, we have investigated the role of deuterium in mitochondrial dysfunction, and the potential role of amyloidogenic proteins in sequestering deuterium within plaque deposits, both in the brain and in the pancreas

HIF1/2 activation in VHL-deficient ccRCC cells caused lipid droplet accumulation in an ACSL3-dependent manner 121

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this may allow fibrosis to progress to HCC by modulating various PTMs that increase DNA adducts of liver cells [9, 331]

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