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It is injected subcutaneously, usually into the abdomen or thigh, and the dose may be adjusted based on your treatment plan

Combined antiangiogenic and antiPD-L1 therapy stimulates tumor immunity through HEV formation

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Mechanistically, TXNIP antagonizes Trx1 by forming a disulfide bond with its Cys32 residue, leading to Trx1 inactivation and increased ROS levels, thereby facilitating lipid peroxidation and ferroptotic signaling