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Ferroptosis is a form of iron-regulated cell death, which is distinguished from other traditional cell death programs by its morphological (shrunken mitochondria with increased membrane density), biochemical (accumulation of free iron and toxic lipid peroxidation, depletion of intracellular antioxidants), and genetic (involvement of many specific genes) features [19]

There is evidence that a loss of the intestinal barrier function contributes to MASLD (Rahman et al., 2016)

Tissue-specific regulation of iron metabolism and heme synthesis: distinct control mechanisms in erythroid cells

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