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However, the receptor-signaling description of BDNF in PD research remains incompleteexisting studies often overlook the functional crosstalk between TrkB and p75NTR, as well as the impact of TrkB subtype diversity (full-length TrkB.TK + and truncated TrkB.T1) on signal output in dopaminergic neurons [59]

The most remarkable finding in the current study, is that all treatments (LC, LCLN, QT, and QTLN) significantly upregulated the expression of all studied steroidogenic genes, thereby significantly reducing the deleterious effects of ATZ on steroidogenesis