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Moreover, Wnt/-catenin activation in TECs induces cellular senescence [33] and mitochondrial dysfunction characterized by mitochondrial loss, increased fragmentation, and elevated reactive oxygen species (ROS) production [34], all of which exacerbate kidney fibrosis

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doi: 10.1016/j.msec.2019.109803

These findings suggest that 4-HNE covalently modifies essential DNA repair enzymes, thereby diminishing the DNA repair capacity of cardiomyocytes and potentially worsening cellular injury and functional deterioration (Pan et al., 2020)