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Importantly, N -acetyl cysteine (NAC) and vitamin C lowered DSBs by 43 and 25% respectively, which was significantly more than any of the other agents studied [9]

However, hyperuricemia plays a role in promoting inflammation, oxidative stress, and endothelial dysfunction underscores its potential contribution to disease pathogenesis

Experimental antioxidant biotherapy for protection of the vascular wall by modified forms of superoxide dismutase and catalase

However, there are conditions and risk factors that can compromise the endogenous production of glutathione by our liver and which are independent of our age