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Oxidative stress and mitochondrial damage in neurodegenerative diseases: From molecular mechanisms to targeted therapies

Crohns disease pathobiont adherent-invasive E coli disrupts epithelial mitochondrial networks with implications for gut permeability

It is a 46-residue peptide built entirely from D-amino acids in a reversed sequence, a design that confers high resistance to proteolytic degradation while preserving the spatial topology needed to engage its target

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Additionally, improving mitochondrial function, enhancing mitophagy, or preventing mitochondrial fragmentation could reduce mtDNA release and subsequent inflammatory signaling