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Mackman, N., Tilley, R

PPAR-gamma agonists inhibit profibrotic phenotypes in human lung fibroblasts and bleomycin-induced pulmonary fibrosis

For example, some chromatin regions that were originally in an open state and available for transcription factor binding may become tight under the influence of the TME, making it difficult for genes related to antitumor immunity to be transcriptionally activated