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Thus, autosomal dominant mutations in the PRKAG2 gene in humans lead to cardiac hypertrophy, ventricular pre-excitationWolff-Parkinson-White syndrome (WPW), a progressive conduction system disease, and vacuolar glycogen accumulation in cardiomyocytes ( PRKAG2 R302Q mutation is associated with cardiac bioenergetic and metabolic deficits, thus causing cardiac dysfunction

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