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Overproduction of ROS can induce cell death via signaling pathways such as autophagy and apoptosis, resulting in oxidative stress

H., Liao, H

Its central melanocortin receptor profile has been the subject of in vitro and ex vivo research examining hypothalamic signaling pathways and neuropeptide receptor pharmacology

The drug also ameliorates mitochondrial dysfunction [5], restores synaptic transmission [6] and exerts protective effects against neuroinflammation [7]

Compared with free SCU, it prolongs the in vitro release spectrum and blood circulation duration of SCU, increases SCU levels in ischemic brain tissue, and significantly reduces cerebral infarction volume