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In turn, sleep deprivation leads to an upregulation of proapoptotic and proinflammatory markers such as the apoptotic chromatin condensation inducer 1 (Acin1), heat shock protein family E member 1 (HSPE1), and golgin subfamily A member 3 (GOLGA3), thus indicating an overall beneficial function of sleep for myelins maintenance and turnover (Vivo and Bellesi, 2019)

doi: 10.1111/j.1365-313X.2007.03280.x

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Similarly, the affinity of cystathionine for CSE is greater than that of cysteine, favouring the generation of cysteine as opposed to H 2 S

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