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[DOI] [PubMed] [Google Scholar] 22.Gurung M., Li Z., You H., Rodrigues R., Jump D.B., Morgun A., Shulzhenko N

major surgery can blunt this metabolic flexibility, leading to T-cell exhaustion or apoptosis (22, 23)

Early controlled release of peroxisome proliferator-activated receptor / agonist GW501516 improves diabetic wound healing through redox modulation of wound microenvironment

Another experiment confirmed that H 2 S (as potentially formed by GSH-mediated GSSH reduction) is unlikely to be involved in radical scavenging: Na 2 S inhibited resorufin formation only weakly (Extended Data Fig

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