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It is therefore tempting to speculate that defects in the interaction between SOD1, Rac1, and alsin 2 influence redox signaling at the endosomal level

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On the other hand, an elevated concentration of homocysteine, and its metabolites such as homocysteine thiolactone and N-homocysteinyllysine result in health problems and has been indicated as an independent risk factor for cardiovascular disease and accelerated atherosclerosis

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