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214 In Budd-Chiari syndrome patients, mice transplanted with patient flora developed liver injury and gut flora dysbiosis, indicating that flora dysbiosis may lead to the disease through immune dysregulation

This confirmed the accumulation of endogenous iron ions and replenishment of exogenous iron ions, ensuring an enhanced ferroptosis therapeutic effect

Additionally, NRF2 activators such as bardoxolone methyl may enhance cellular antioxidant defenses, reducing ferroptosis-mediated skin damage

Another pathway that allows deglutathionylation is independent of GSH and involves thioredoxin (Trx), a small, ubiquitous redox protein

Taylor CG