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Already in the 90s, early studies identified key regulators of protein synthesis, such as S6K1 and 4E-BP1, to be responsive to AA sufficiency, thus linking AA to mTORC1 activation (Hara et al., 1998)

These findings align with prior studies indicating that AAA pathogenesis involves pronounced recruitment of innate and adaptive immune cells, contributing to tissue degradation and remodeling[42, 48]

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Baseline characteristics The mean age of each group was 57.89.4 years in Group 1, and 59.38.7 years in Group 2, respectively (p=0.439)