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Wilson's disease The protein, ATP7B, is responsible for the excretion of hepatic copper into the biliary tract, and its impairment in Wilson's disease results in an increased concentration of 'free' copper (i.e., not bound to the copper-carrying protein, ceruloplasmin) in blood, an increased excretion of copper in the urine (hypercupriuria), the deposition of copper in part of the cornea (forming Kayser-Fleischer rings), and the accumulation of copper in the liver and brain (106)

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Lopez-BarbosaNGarciaJGCifuentesJCastroLMVargasFOstosCet alMultifunctional magnetite nanoparticles to enable delivery of siRNA for the potential treatment of Alzheimers