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-synuclein toxicity in neurodegeneration: mechanism and therapeutic strategies

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For all procedures, find buffer compositions in Supplemental Table 1

Furthermore, increased expression of heme oxygenase-1 (HO-1) in microglia and loss of copper chaperone protein (CP) activity in astrocytes contribute to iron deposition in the central nervous system (CNS) and neuronal death [182]

The experiments indicated that MCPyV-positive MCC cells could be eliminated by artesunate through dysregulation of lipid peroxides and eventually triggering a ferroptotic process