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Patients with decompensated MASH cirrhosis were at an increased risk of all-cause mortality (HR: 52.24, 95% CI 24.28,112.40) followed by compensated MASH cirrhosis (HR: 13.62, 95% CI 6.13,30.29) compared to MASH without cirrhosis (Figure 1c)

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This extended receptor activation is therapeutically important: it mimics the sustained reproductive hormone response needed to trigger follicular development

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