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Furthermore, our own studies have shown that pharmacological targeting of NK-1R with the antagonist L703,606 can not only prevent the development of damaging inflammation due to streptococcal CNS infection when administered prophylactically, but can also reverse infection-associated gliosis and demyelination when delivered therapeutically without increasing CNS bacterial burden (Chauhan et al., 2011)

Lgr5 + intestinal stem cells have been identified as playing a crucial role in regenerative repair of radiation-induced intestinal injury [263], but their involvement in fibrosis remains unclear