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doi:10.1146/annurev-biochem-051110-133601

Nonetheless, an exhaustive list of reviews, hypotheses, and experimental work examining mechanisms including, but not limited to, neuroinflammation [2225], mitochondrial quality control [26, 27], mitochondrial DNA content [28, 29], and endoplasmic reticulum stress [30] have been presented as causes responsible for the activation of Neuro-PASC [3137]

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Whether combining ferroptosis-related HNC treatments with existing HNC treatments may produce improved outcomes, as well as how the adverse effects of ferroptosis in the treatment of HNC can be avoided, are questions to be addressed by future research

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