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Dysfunctional mitochondria can release and misplace mitochondrial components, which serve as DAMPs and induce inflammation via PRRs, through multiple mechanisms (88)

On the basis of recent studies suggesting that AAA-derived metabolites could be potent aryl hydrocarbon receptor (AhR) ligands in the context of host immune system function 16 , mucosal reactivity 17 and oxidative stress defense 18 , we use a Gaussia luciferase (GLuc) reporter system to monitor the ability of the identified metabolites to activate the AhR

doi: 10.3389/fmolb.2021.832022 136 HassirianNKarimiEOskoueianE

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Severe reactive astrocytes precipitate pathological hallmarks of Alzheimers disease via H 2 O 2 - production