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Under oxidative stress, excessive Ca 2+ influx into mitochondria exacerbates mPTP activation and m loss ( 2+ dysregulation converge to drive mitochondrial collapse, ATP depletion, and skeletal myocyte apoptosis or necrosis-ultimately contributing to the pathogenesis of muscle atrophy ( 5.2.2 Mitochondrial dysfunction exacerbates ROS production: a vicious cycle Mitochondrial dysfunction both amplifies ROS generation and impairs antioxidant defense, establishing a self-perpetuating cycle of oxidative injury and organelle deterioration (Yazdani et al., 2023)

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doi: 10.1016/j.clim.2016.08.019

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The Confidence Tiers We Use in This Review To keep this grounded, each item is labeled by evidence strength and real-world usefulness: Tier 1 (Strong): Consistent benefits across multiple studies and real-world use