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Conclusion In conclusion, this review provides a comprehensive summary of recent molecular research shedding light on the complex mechanisms through which HIV-1-related factorssuch as viral proteins and RNAsmediate microglial activation, leading to neuroinflammation and the development of NeuroHIV

GSS -deficient patients exhibit significant metabolic consequences since the conversion of accumulated -glutamylcysteine to 5-oxo-proline can cause severe metabolic acidosis, hemolytic anemia, and central nervous system damage [24, 25]