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However, the gradual depletion of stores means that deficiency develops insidiously

5 miRNAs associated with anti-inflammatory M2-type microglia in PD 5.1 miR-124 The primary mechanism through which extracellular vesicles (EVs) derived from mesenchymal stem cells improve neuroinflammation and neurological function involves the direct modulation of microglial activation in conditions associated with PD (Yao et al., 2018)

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Therefore, similar to p53, BAP1 suppresses tumor development at least partly by inducing ferroptosis upon SLC7A11 repression (Fig

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