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This biochemical mechanism explains: Cyclophosphamide Cyclophosphamide is an anti-cancer drug

(2022) expanded on these findings, showing that a water-soluble BSP isolate not only enhanced macrophage activity but also increased the production of ROS and elevated the expression of key pro-inflammatory cytokines, including interleukin-1 (IL-1) and tumor necrosis factor- (TNF-)

Animal models for human BPD use several approaches to replicate the major findings of alveolar simplification and abnormal vascular remodeling in the murine lung including postnatal hyperoxia exposure in the term mouse [14,15]

This pro-ferroptotic role of NO provides the rationale for using NO-releasing drugs to induce ferroptotic cell death in CRC (Tables 2, 3)

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