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The oxidative stress is a major cause for the exhibiting the cardiotoxicity, involved the generation of higher amounts of ROS cause the cellar alterations and damage are referred to as oxidative stress

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We hypothesize that the role of HPV-16 E7 is to establish a subset of GSTP1 molecules that is inaccessible to oxidative attack, thus creating a reservoir of reduced monomeric GSTP1

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