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Chronic aspartame intake causes changes in the trans-sulphuration pathway, glutathione depletion and liver damage in mice

Loss and accuracy functions were used to evaluate the ANN model and monitor the error during the training process

Most commonly, oligodendrocyte differentiation abnormalities are associated with disruption of oxidative stress phenomena associated with excitotoxicity, mediated by glutamatergic metabotropic receptors in large amounts in oligodendrocyte precursors [22, 137]

Lack of glutathione peroxidase-1 exacerbates a beta-mediated neurotoxicity in cortical neurons

When combined, these substances can lead to stomach irritation and may result in internal bleeding, ulcers, and liver damage