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Therefore, the scope of our article is to summarize what is known about the features and pathophysiology of the most prevalent IDDs and to explore the implication of glia in neuroinflammation, demyelination, neurodegeneration and clinical treatment throughout the course of disease

TLR4 binding to ligand molecules induces conformational and charge distribution changes that activate the cytoplasmic TIR region, subsequently initiating downstream pathways via either the myeloid differentiation factor 88 (MyD88)-dependent or MyD88-independent pathways (188)

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