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During the activation phase, stimuli such as Nigericin (113) prompted NLRP3 to oligomerize through the NACHT structural domain, recruit apoptosis-associated speck-like protein containing a CARD (ASC) and NIMA-associated kinase 7 (NEK7), and promote ASC oligomerization to form macromolecular foci of specks, which in turn led to pro-caspase-1 recruitment and activation, and assembly to form NLRP3 inflammatory vesicles (114)

elegans VHL homolog), leading to poly-ubiquitination and proteasomal degradation of HIF-1

Next, well look at how India regulates stem cell therapies and compare this to how other countries do it

10.1016/j.jinorgbio.2015.09.003 58 SteinbrennerH.SiesH

POSTAR3: an updated platform for exploring post-transcriptional regulation coordinated by RNA-binding proteins