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However, MT gene expression largely varies depending on the cancer cell type, and its regulation contributes to protection against DNA damage and apoptosis

From a biochemical perspective, the peptides reported resistance to enzymatic degradation may be attributed to the presence of a disulfide bridge between cysteine residues, along with an N-terminal tyrosine substitution, which potentially supports its structural resilience under experimental conditions, such as in vitro digestion models

escalating a fixed-ratio blend is not the documented response