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As the efficacy of single-agent metabolic inhibitors is often limited by metabolic plasticity, developing rational combination therapies based on the coordinated targeting of oncogenic signaling and its downstream metabolic dependencies holds promise for systematically overcoming resistance and achieving more durable therapeutic benefits in tumors with specific genetic backgrounds

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10.17992/lbl.2024.03.785 16

Manganese-induced miR-125b-2-3p promotes anxiety-like behavior via TFR1-mediated ferroptosis

Vascular-promoting therapy reduced tumor growth and progression by improving chemotherapy efficacy