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Moreover, GH signaling to elicit IGF1 gene transcription is blocked in the absence of appropriate insulin signaling, a phenomenon also known as un-coupling, and resulting in growth arrest (60, 61, 63, 64) (Figure 3)

Evidence position Predominantly laboratory and animal research, with very limited human data

Sleep-enhancing peptides exert systemic effects by acting on circadian regulators (e.g., CLOCK, BMAL1), restoring pineal-mitochondrial signaling, and enhancing endogenous GH release (Table 3)