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doi: 10.1001/jamaoncol.2015.1377 140 MiyakawaY.OtsukaM.ShibataC.SeimiyaT.YamamotoK.IshibashiR.et al

455 Future research should elucidate how aberrant sensory afferent activity interacts with maladaptive CNS plasticity to initiate and sustain neuropathic pain

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NFAT5 repressed NIK/NF-B2 pathway via ubiquitination and degradation of NIK by Akt1/cIAP1/2 We used the STRING database to identify the mediator molecule between NFAT5 and NIK/NF-B2

Its pathological features include high inflammatory and immune response, oxidative stress, defective antioxidant defense system, altered protein homeostasis, cellular senescence, and apoptosis