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In an in vivo study utilizing TIGAR conditional knockout mice, TIGAR loss impaired tumor formation in intestinal colon cancer generated by the loss of the tumor suppressor APC

A., Bader Lange M

MGO is known to be increased in endothelial cells during hyperglycemic events due to the insulin-independent uptake of glucose [22]

The blot was normalized against -actin levels

p53-reactive T cells are associated with clinical benefit in patients with platinum-resistant epithelial ovarian cancer after treatment with a p53 vaccine and gemcitabine chemotherapy