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JNK1/2 (c-Jun N-terminal kinases) show slight inhibition with reduced phosphorylation, though effects are less pronounced than p38 MAPK pathway changes

This cautious approach mirrors the careful planning required for surgical procedures to ensure optimal outcomes

GHK-Cu exerts its effects primarily through gene expression modulation, influencing approximately 31% of the human genome by upregulating genes involved in antioxidant responses, DNA repair, protein metabolism, and cellular growth while downregulating inflammatory mediators, fibrinogen synthesis, and pathological processes including cancer metastasis