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Neutrophils can express Gasdermin D (GSDMD) and maintain their ability to release IL-1 (a pro-inflammatory cytokine) through a GSDMD-dependent pathway, thereby promoting inflammatory responses and immune responses without cell death (111)

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The chronic administration of D-gal induces changes that resemble natural aging in animals, such as cognitive dysfunction, oxidative stress and neurodegradation

Resilience to chronic stress is mediated by hippocampal brain-derived neurotrophic factor