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Abstract Background Arterial or venous thromboembolic events are responsible for one-fourth of all deaths worldwide

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doi: 10.1007/s00604-018-2830-6

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The intracellular Fe 2+ can be exported by iron-efflux protein ferroportin (FPN1, also known as solute carrier family 40 member 1, SLC40A1) and then be reoxidized to Fe 3+ [35]

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this single delay is medically insignificant

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