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In TNF-stimulated mouse cardiac muscle cells, sodium hydrosulfide ameliorated the impaired mitochondrial respiration and ATP production/synthesis, and attenuated excess oxidative stress, which might be due to the enhanced expression of metabolic indices such as CPT1

Impaired Mitochondrial Function of Alveolar Macrophages in Carbon Nanotube-Induced Chronic Pulmonary Granulomatous Disease

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