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Unlike in early APAP poisoning, where NAC functions primarily as a glutathione precursor to detoxify N-acetyl-p-benzoquinone imine (NAPQI), in FHF, NAC is believed to exert additional hepatoprotective and systemic benefits

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In 2016, the cereblon-based MGD CC-885 was reported to be capable of inducing the degradation of the transcription factor GSPT1, and thus presenting a potent preclinical anticancer activity, more specifically an antiproliferative effect in AML cells 210

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