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51,53 Collectively, these findings suggest that both canonical and noncanonical NF-B pathways are involved in the generation and effector functions of inflammatory T cells, although they differ in mechanisms of activation and function

These findings could enhance our understanding and therapeutic targeting of the metabolic role of the PNPLA3 in immunometabolism

Thomas AM, Atkinson TJ

Mechanisms relevant to psoriasis: Reduces NF-B activity (a key inflammatory pathway in psoriasis) Decreases pro-inflammatory cytokines Promotes collagen synthesis and skin remodeling Activates wound healing gene expression Anti-oxidant properties reduce oxidative stress in lesions Research highlight: GHK-Cu has been shown to reset gene expression patterns associated with aging and inflammation

Suppression of persistent atrial fibrillation by genetic knockdown of caspase 3: a pre-clinical pilot study