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The various roles of fatty acids

While the dithiol mechanism for EcGrx1 and EcRNR or EcPR can be explained by specific protein-protein interactions (Fig

Western blotting showed that both SLC7A11 and GPX4, which activate inhibitory signals, were expressed at higher levels in the GSH-treated group than in the control group, similar to the expression pattern of the adipogenesis-related marker PPAR (Fig