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We have identified changes in key antioxidant enzymes that may shed light on the mechanism of the protective effects of ALA

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Currently, different treatment options are available, as shown in Figure 1C, including pharmacological treatment, but every treatment strategy has its own limiting factors

Keywords: GHK peptide, fibroblasts, myofibroblasts, senescence, fibrosis, idiopathic pulmonary fibrosis The loss of cellular homeostasis in fibrosis is characterized by the accumulation of activated fibroblasts, known as myofibroblasts [1], which are responsible for excessive extracellular matrix deposition and tissue remodeling

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