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High levels of NRF2 sensitize temozolomide-resistant glioblastoma cells to ferroptosis via ABCC1/MRP1 upregulation

The negligible increment of ascorbate-glutathione content, ascorbate-glutathione redox and ascorbate-glutathione cycle enzymes activities and dramatic increment in malondialdehyde (MDA), hydrogen peroxide (H 2 O 2 ) and EL were observed in the sensitive genotype VA15

0.4%, p=0.09)

This creates an immediate antioxidant surge in the blood, but not necessarily inside the cells where glutathione is actually required to function

Furthermore, endothelial cells in the TME activate the expression of the Notch1 receptor (N1ICD) and facilitate peritoneal metastasis