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When an individual has an MTHFR polymorphism, their body struggles to efficiently convert synthetic cyanocobalamin (the cheap form of B12 found in most standard supplements and fortified foods) into the active methylcobalamin required by the cells

Functionally this manifests as directional cell migration - endothelial cells, keratinocytes, fibroblasts, and cardiac progenitors all migrate more efficiently in TB-500-treated models (Sosne 2002, 2010

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