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Some studies in animal models raised concerns about potential cancer risks with prolonged use, leading to its discontinuation in pharmaceutical research

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Thus, the alteration of GABA-glutamate equilibrium resulted in a sustained high GABA/glutamate ratio in JEV-infected brain, which is secondary to the aberrant glutamine-glutamate metabolism described before

Fonseca MI, Chu S-H, Hernandez MX, Fang MJ, Modarresi L, Selvan P, MacGregor GR and Tenner AJ (2017) Cell-specific deletion of C1qa identifies microglia as the dominant source of C1q in mouse brain