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The underlying pathophysiology of facial synkinesis is likely multifactorial, although the predominant mechanism appears to be aberrant regeneration of facial nerve fibres to the facial muscle groups following facial nerve injury [4]

Protein expression We used the Bac-to-Bac baculovirus system (Thermo Fisher) in Spodoptera frugiperda ( sf9 ) cells for expression

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The activation of IL-12p40 has previously been attributed to hypoxia-inducible factor-1, a key regulator of mucosal inflammation by controlling Th1/Th17 response [69]

0.5 mL/kg for Group 3 and 1.5 mL/kg for Group 4