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These initial studies in CA1 inhibitory neurons also demonstrate that plasticity rules and mechanisms in interneurons can be: (a) different from those known for pyramidal cells, e.g., NMDA-independent (Perez et al., 2001

10.3233/JAD-180176 526 ZoladzP

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However, the intricate molecular mechanisms underlying this observation, whether through direct interaction with iNOS or by counteracting the downstream effects of excessive NO, remain to be elucidated in detail