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In conclusion, VEGF participates to CD8 + T cell exclusion without affecting Treg, promoting immunosuppression [28]

These interactions can trigger a series of immunoregulatory responses, involving both innate and adaptive immune systems and including cross-presentation of antigens, activation of CD 8 + T cells and CD 4 + T cells, phagocytosis, complement-mediated antibody-dependent cellular cytotoxicity (ADCC) and complement-dependent cytotoxicity (CDC)

By increasing glycine availability at the NMDAR co-agonist site, GlyT1 inhibition may improve receptor function, restore interneuron activity, and rebalance excitation and inhibition within affected neural circuits

doi:10.1007/BF02837864

These studies should also evaluate the long-term safety and efficacy of these therapies, particularly in the context of ICI and emerging treatments [148]