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The most significantly downregulated protein following treatment of cells with either PROTAC 1a or 2c was mitochondrial Acetyl-CoA acetyltransferase (ACAT1), which catalyzes the last step of the mitochondrial beta-oxidation pathway

We also compared the change in plasma GPT and GOT levels in the wild-type and PDI fl/fl Alb-cre mice following different treatments

The safety profile was however good as also the objective response

To view a copy of this licence, visit About this article Cite this article Sasula, MJ., Held, A.T.J., Schefczyk, S

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