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Inflammation in atherosclerosis: pathophysiology and mechanisms

Management includes dose reduction or discontinuation of the drug

Studies have suggested that the uptake of HMCD complexes, including MHI-148-HSA and genistein-IR-783 conjugates, could be mediated by micropinocytosis and lipid-raft endocytosis ( Figure 2 ) (58, 74)

Bibcode:2000PNAS...97.8841N

Compared with diabetic rats, the left ventricular hemodynamic parameters and myocardial ultrastructure changes (myocardial hypertrophy and fibrosis) are obviously normalized in diabetic animals treated with NaHS, which may be associated with upregulation of cardiac myosin light chain kinase (MLCK) and downregulation of serum creatine kinase MB isozyme (CK-MB) (Yang et al., 2016a)